Summary
This study exposed young female mice to one day of high heat and humidity to assess cardiac consequences of environmental heat stress. Core temperature rose by about 2.1 °C and the heart showed edema, ventricular muscle-cell vacuolation, mitochondrial remodeling, increased autophagy, and endoplasmic-reticulum stress, demonstrating that a single prolonged exposure can injure the myocardium. UID UCT-2112 temperature microchips supported thermal monitoring.
Summary written by UID from the published work. Read the original publication for the authors' abstract and full methods.