Summary
This study established a mouse model of persistent neurologic effects after mild SARS-CoV-2 infection. Recovered male and female K18-hACE2 mice developed lung fibrosis, elevated kinin B1 receptor and inflammatory markers in the lungs and brain, greater anxiety, and less exploratory behavior, implicating sustained B1-receptor signaling in long-term neuroinflammation and cognitive symptoms. UID UCT-2112 temperature microchips supported infection and recovery monitoring.
Summary written by UID from the published work. Read the original publication for the authors' abstract and full methods.